Accès gratuit
Numéro |
Biologie Aujourd'hui
Volume 205, Numéro 1, 2011
|
|
---|---|---|
Page(s) | 47 - 52 | |
Section | Plasticité du phénotype cellulaire : entre régénération et tumorigenèse | |
DOI | https://doi.org/10.1051/jbio/2011001 | |
Publié en ligne | 19 avril 2011 |
- Ansieau S., Bastid J., Doreau A., Morel A.P., Bouchet B.P., Thomas C., Fauvet F., Puisieux I., Doglioni C., Piccinin S., Maestro R., Voeltzel T., Selmi A., Valsesia-Wittmann S., Caron de F.C., Puisieux A., Induction of EMT by twist proteins as a collateral effect of tumor-promoting inactivation of premature senescence. Cancer Cell, 2008a, 14, 79–89. [CrossRef] [PubMed] [Google Scholar]
- Ansieau S., Hinkal G., Thomas C., Bastid J., Puisieux A., Early origin of cancer metastases: dissemination and evolution of premalignant cells. Cell Cycle, 2008b, 7, 3659–3663. [CrossRef] [PubMed] [Google Scholar]
- Ansieau S., Morel A.P., Hinkal G., Bastid J., Puisieux A., TWISTing an embryonic transcription factor into an oncoprotein. Oncogene, 2010, 29, 3173–3184. [CrossRef] [PubMed] [Google Scholar]
- Bartkova J., Horejsi Z., Koed K., Kramer A., Tort F., Zieger K., Guldberg P., Sehested M., Nesland J.M., Lukas C., Orntoft T., Lukas J., Bartek J., DNA damage response as a candidate anti-cancer barrier in early human tumorigenesis. Nature, 2005, 434, 864–870. [CrossRef] [PubMed] [Google Scholar]
- Bartkova J., Rezaei N., Liontos M., Karakaidos P., Kletsas D., Issaeva N., Vassiliou L.V., Kolettas E., Niforou K., Zoumpourlis V.C., Takaoka M., Nakagawa H., Tort F., Fugger K., Johansson F., Sehested M., Andersen C.L., Dyrskjot L., Orntoft T., Lukas J., Kittas C., Helleday T., Halazonetis T.D., Bartek J., Gorgoulis V.G., Oncogene-induced senescence is part of the tumorigenesis barrier imposed by DNA damage checkpoints. Nature, 2006, 444, 633–637. [CrossRef] [PubMed] [Google Scholar]
- Battula V.L., Evans K.W., Hollier B.G., Shi Y., Marini F.C., Ayyanan A., Wang R.Y., Brisken C., Guerra R., Andreeff M., Mani S.A., Epithelial-mesenchymal transition-derived cells exhibit multilineage differentiation potential similar to mesenchymal stem cells. Stem Cells, 2010, 28, 1435–1445. [CrossRef] [PubMed] [Google Scholar]
- Braig M., Lee S., Loddenkemper C., Rudolph C., Peters A.H., Schlegelberger B., Stein H., Dorken B., Jenuwein T., Schmitt C.A., Oncogene-induced senescence as an initial barrier in lymphoma development. Nature, 2005, 436, 660–665. [CrossRef] [PubMed] [Google Scholar]
- Brosh R., Rotter V., When mutants gain new powers: news from the mutant p53 field. Nat Rev Cancer, 2009, 9, 701–713. [PubMed] [Google Scholar]
- Collado M., Gil J., Efeyan A., Guerra C., Schuhmacher A.J., Barradas M., Benguria A., Zaballos A., Flores J.M., Barbacid M., Beach D., Serrano M., Tumour biology: senescence in premalignant tumours. Nature, 2005, 436, 642. [CrossRef] [PubMed] [Google Scholar]
- Creighton C.J., Li X., Landis M., Dixon J.M., Neumeister V.M., Sjolund A., Rimm D.L., Wong H., Rodriguez A., Herschkowitz J.I., Fan C., Zhang X., He X., Pavlick A., Gutierrez M.C., Renshaw L., Larionov A.A., Faratian D., Hilsenbeck S.G., Perou C.M., Lewis M.T., Rosen J.M., Chang J.C., Residual breast cancers after conventional therapy display mesenchymal as well as tumor-initiating features. Proc Natl Acad Sci USA, 2009, 106, 13820–13825. [CrossRef] [Google Scholar]
- Evan G.I., Wyllie A.H., Gilbert C.S., Littlewood T.D., Land H., Brooks M., Waters C.M., Penn L.Z., Hancock D.C., Induction of apoptosis in fibroblasts by c-myc protein. Cell, 1992, 69, 119–128. [CrossRef] [PubMed] [Google Scholar]
- Gupta P.B., Onder T.T., Jiang G., Tao K., Kuperwasser C., Weinberg R.A., Lander E.S., Identification of selective inhibitors of cancer stem cells by high-throughput screening. Cell, 2009, 138, 645–659. [CrossRef] [PubMed] [Google Scholar]
- Hermeking H., Eick D., Mediation of c-Myc-induced apoptosis by p53. Science, 1994, 265, 2091–2093. [CrossRef] [PubMed] [Google Scholar]
- Husemann Y., Geigl J.B., Schubert F., Musiani P., Meyer M., Burghart E., Forni G., Eils R., Fehm T., Riethmuller G., Klein C.A., Systemic spread is an early step in breast cancer. Cancer Cell, 2008, 13, 58–68. [CrossRef] [PubMed] [Google Scholar]
- Junttila M.R., Evan G.I., p53–a Jack of all trades but master of none. Nat Rev Cancer, 2009, 9, 821–829. [CrossRef] [PubMed] [Google Scholar]
- Klein C.A., Cancer. The metastasis cascade. Science, 2008, 321, 1785–1787. [CrossRef] [PubMed] [Google Scholar]
- Maestro R., Dei Tos A.P., Hamamori Y., Krasnokutsky S., Sartorelli V., Kedes L., Doglioni C., Beach D.H., Hannon G.J., Twist is a potential oncogene that inhibits apoptosis. Genes Dev, 1999, 13, 2207–2217. [CrossRef] [PubMed] [Google Scholar]
- Mani S.A., Guo W., Liao M.J., Eaton E.N., Ayyanan A., Zhou A.Y., Brooks M., Reinhard F., Zhang C.C., Shipitsin M., Campbell L.L., Polyak K., Brisken C., Yang J., Weinberg R.A., The epithelial-mesenchymal transition generates cells with properties of stem cells. Cell, 2008, 133, 704–715. [CrossRef] [PubMed] [Google Scholar]
- Morel A.P., Lièvre M., Thomas C., Hinkal G., Ansieau S., Puisieux A., Generation of breast cancer stem cells through epithelial-mesenchymal transition. PLoS One, 2008, 3, e2888. [CrossRef] [PubMed] [Google Scholar]
- Ohashi S., Natsuizaka M., Wong G.S., Michaylira C.Z., Grugan K.D., Stairs D.B., Kalabis J., Vega M.E., Kalman R.A., Nakagawa M., Klein-Szanto A.J., Herlyn M., Diehl J.A., Rustgi A.K., Nakagawa H., Epidermal growth factor receptor and mutant p53 expand an esophageal cellular subpopulation capable of epithelial-to-mesenchymal transition through ZEB transcription factors1. Cancer Res, 2010, 70, 4174–4184. [CrossRef] [PubMed] [Google Scholar]
- Peinado H., Olmeda D., Cano A., Snail, Zeb and bHLH factors in tumour progression: an alliance against the epithelial phenotype? Nat Rev Cancer, 2008, 7, 415–428. [Google Scholar]
- Schardt J.A., Meyer M., Hartmann C.H., Schubert F., Schmidt-Kittler O., Fuhrmann C., Polzer B., Petronio M., Eils R., Klein C.A., Genomic analysis of single cytokeratin-positive cells from bone marrow reveals early mutational events in breast cancer. Cancer Cell, 2005, 8, 227–239. [Google Scholar]
- Serrano M., Lin A.W., McCurrach M.E., Beach D., Lowe S.W., Oncogenic ras provokes premature cell senescence associated with accumulation of p53 and p16INK4a. Cell, 1997, 88, 593–602. [CrossRef] [PubMed] [Google Scholar]
- Stoecklein N.H., Hosch S.B., Bezler M., Stern F., Hartmann C.H., Vay C., Siegmund A., Scheunemann P., Schurr P., Knoefel W.T., Verde P.E., Reichelt U., Erbersdobler A., Grau R., Ullrich A., Izbicki J.R., Klein C.A., Direct genetic analysis of single disseminated cancer cells for prediction of outcome and therapy selection in esophageal cancer. Cancer Cell, 2008, 13, 441–453. [CrossRef] [PubMed] [Google Scholar]
- Thiéry J.P., Acloque H., Huang R.Y., Nieto M.A., Epithelial-mesenchymal transitions in development and disease. Cell, 2009, 139, 871–890. [CrossRef] [PubMed] [Google Scholar]
- Thisse B., el Messal M., Perrin-Schmitt F., The twist gene: isolation of a Drosophila zygotic gene necessary for the establishment of dorsoventral pattern. Nucleic Acids Res, 1987, 15, 3439–3453. [CrossRef] [PubMed] [Google Scholar]
- Valsesia-Wittmann S., Magdeleine M., Dupasquier S., Garin E., Jallas A.C., Combaret V., Krause A., Leissner P., Puisieux A., Oncogenic cooperation between H-Twist and N-Myc overrides failsafe programs in cancer cells. Cancer Cell, 2004, 6, 625–630. [CrossRef] [PubMed] [Google Scholar]
- Vesuna F., Lisok A., Kimble B., Raman V., Twist modulates breast cancer stem cells by transcriptional regulation of CD24 expression1. Neoplasia, 2009, 11, 1318–1328. [PubMed] [Google Scholar]
- Wang Z., Li Y., Ahmad A., Azmi A.S., Kong D., Banerjee S., Sarkar F.H., Targeting miRNAs involved in cancer stem cell and EMT regulation: An emerging concept in overcoming drug resistance. Drug Resist Update, 2010, In press. [Google Scholar]
- Yang J., Mani S.A., Donaher J.L., Ramaswamy S., Itzykson R.A., Come C., Savagner P., Gitelman I., Richardson A., Weinberg R.A., Twist, a master regulator of morphogenesis, plays an essential role in tumor metastasis. Cell, 2004, 117, 927–939. [CrossRef] [PubMed] [Google Scholar]
Les statistiques affichées correspondent au cumul d'une part des vues des résumés de l'article et d'autre part des vues et téléchargements de l'article plein-texte (PDF, Full-HTML, ePub... selon les formats disponibles) sur la platefome Vision4Press.
Les statistiques sont disponibles avec un délai de 48 à 96 heures et sont mises à jour quotidiennement en semaine.
Le chargement des statistiques peut être long.