Accès gratuit
Numéro
Biologie Aujourd'hui
Volume 205, Numéro 3, 2011
Page(s) 139 - 146
Section Cytosquelette et pathologies / Cytoskeleton and pathologies
DOI https://doi.org/10.1051/jbio/2011015
Publié en ligne 11 octobre 2011
  • Colucci-Guyon E., Portier M.M., Dunia I., Paulin D., Pournin S., Babinet C., Mice lacking vimentin develop and reproduce without an obvious phenotype. Cell, 1994, 79, 679–694. [CrossRef] [PubMed] [Google Scholar]
  • Coulombe P.A., Kerns M.L., Fuchs E., Epidermolysis bullosa simplex: a paradigm for disorders of tissue fragility. J Clin Invest, 2009, 119, 1784–1793. [CrossRef] [PubMed] [Google Scholar]
  • Elder G.A., Friedrich V.L., Jr., Bosco, P., Kang, C., Gourov, A., Tu, P.H., Lee, V.M., Lazzarini, R.A., Absence of the mid-sized neurofilament subunit decreases axonal calibers, levels of light neurofilament (NF-L), and neurofilament content. J Cell Biol, 1998, 141, 727–739. [CrossRef] [PubMed] [Google Scholar]
  • Goldfarb L.G., Dalakas M.C., Tragedy in a heartbeat: malfunctioning desmin causes skeletal and cardiac muscle disease. J Clin Invest, 2009, 119, 1806–1813. [CrossRef] [PubMed] [Google Scholar]
  • Herrmann H., Strelkov S.V., Burkhard P., Aebi U., Intermediate filaments: primary determinants of cell architecture and plasticity. J Clin Invest, 2009, 119, 1772–1783. [Google Scholar]
  • Ishikawa H., Bischoff R., Holtzer H., Mitosis and intermediate-sized filaments in developing skeletal muscle. J Cell Biol, 1968, 38, 538–555. [CrossRef] [PubMed] [Google Scholar]
  • Larivière R.C., Nguyen M.D., Ribeiro-da-Silva, A., Julien, J.P., Reduced number of unmyelinated sensory axons in peripherin null mice. J Neurochem, 2002, 81, 525–532. [CrossRef] [PubMed] [Google Scholar]
  • Li Z., Colucci-Guyon E., Pinçon-Raymond M., Mericskay M., Pournin S., Paulin D., Babinet C., Cardiovascular lesions and skeletal myopathy in mice lacking desmin. Dev Biol, 1996, 175, 362–366. [CrossRef] [PubMed] [Google Scholar]
  • Liem R.K., Messing A., Dysfunctions of neuronal and glial intermediate filaments in disease. J Clin Invest, 2009, 119, 814–1824. [Google Scholar]
  • McCullagh K.J., Edwards B., Kemp M.W., Giles L.C., Burgess M., Davies K.E., Analysis of skeletal muscle function in the C57BL6/SV129 syncoilin knockout mouse. Mamm Genome, 2008, 19, 339–351. [CrossRef] [PubMed] [Google Scholar]
  • Milner D.J., Weitzer G., Tran D., Bradley A., Capetanaki Y., Disruption of muscle architecture and myocardial degeneration in mice lacking desmin. J Cell Biol, 1996, 134, 1255–1270. [CrossRef] [PubMed] [Google Scholar]
  • Omary M.B., Ku N.O., Strnad P., Hanada S., Toward unraveling the complexity of simple epithelial keratins in human disease. J Clin Invest, 2009, 119, 1794–1805. [CrossRef] [PubMed] [Google Scholar]
  • Oshima R.G., Intermediate filaments: a historical perspective. Exp Cell Res, 2007, 313, 1981–1994. [CrossRef] [PubMed] [Google Scholar]
  • Paulin D., Huet A., Khanamyrian L., Xue Z., Desminopathies in muscle disease. J Pathol, 2004, 204, 418–427. [CrossRef] [PubMed] [Google Scholar]
  • Song S., Landsbury A., Dahm R., Liu Y., Zhang Q., Quinlan R.A., Functions of the intermediate filament cytoskeleton in the eye lens. J Clin Invest, 2009, 119, 1837–1848. [CrossRef] [PubMed] [Google Scholar]
  • Worman H.J., Fong L.G., Muchir A., Young S.G., Laminopathies and the long strange trip from basic cell biology to therapy. J Clin Invest, 2009, 119, 1825–1836. [CrossRef] [PubMed] [Google Scholar]
  • Xue Z.G., Chéraud Y., Brocheriou V., Izmiryan A., Titeux M., Paulin D., Li Z., The mouse synemin gene encodes three intermediate filament proteins generated by alternative exon usage and different open reading frames. Exp Cell Res, 2004, 298, 431–444. [CrossRef] [PubMed] [Google Scholar]
  • Zhang J., Bang M.L., Gokhin D.S., Lu Y., Cui L., Li X., Gu Y., Dalton N.D., Scimia M.C., Peterson K.L., Lieber R.L., Chen J., Syncoilin is required for generating maximum isometric stress in skeletal muscle but dispensable for muscle cytoarchitecture. Am J Physiol Cell Physiol, 2008, 294, C1175–1182. [CrossRef] [PubMed] [Google Scholar]
  • Zhu Q., Lindenbaum M., Levavasseur F., Jacomy H., Julien J.P., Disruption of the NF-H gene increases axonal microtubule content and velocity of neurofilament transport: relief of axonopathy resulting from the toxin beta,beta’-iminodipropionitrile. J Cell Biol, 1998, 143, 183–193. [CrossRef] [PubMed] [Google Scholar]

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